Albarracin CT - Search Results

1

Annexin A1 Preferentially Predicts Poor Prognosis of Basal-Like Breast Cancer Patients by Activating mTOR-S6 Signaling.

Bhardwaj A, Ganesan N, Tachibana K, Rajapakshe K, Albarracin CT, Gunaratne PH, Coarfa C, Bedrosian I. Bhardwaj A, et al. Among authors: albarracin ct. PLoS One. 2015 May 22;10(5):e0127678. doi: 10.1371/journal.pone.0127678. eCollection 2015. PLoS One. 2015. PMID: 26000884 Free PMC article.

2

Expression of annexin A1 in esophageal and esophagogastric junction adenocarcinomas: association with poor outcome.

Wang KL, Wu TT, Resetkova E, Wang H, Correa AM, Hofstetter WL, Swisher SG, Ajani JA, Rashid A, Hamilton SR, Albarracin CT. Wang KL, et al. Among authors: albarracin ct. Clin Cancer Res. 2006 Aug 1;12(15):4598-604. doi: 10.1158/1078-0432.CCR-06-0483. Clin Cancer Res. 2006. PMID: 16899607

3

Expression of epidermal growth factor receptor in esophageal and esophagogastric junction adenocarcinomas: association with poor outcome.

Wang KL, Wu TT, Choi IS, Wang H, Resetkova E, Correa AM, Hofstetter WL, Swisher SG, Ajani JA, Rashid A, Albarracin CT. Wang KL, et al. Among authors: albarracin ct. Cancer. 2007 Feb 15;109(4):658-67. doi: 10.1002/cncr.22445. Cancer. 2007. PMID: 17211865 Free article.

4

MicroRNA-196a targets annexin A1: a microRNA-mediated mechanism of annexin A1 downregulation in cancers.

Luthra R, Singh RR, Luthra MG, Li YX, Hannah C, Romans AM, Barkoh BA, Chen SS, Ensor J, Maru DM, Broaddus RR, Rashid A, Albarracin CT. Luthra R, et al. Among authors: albarracin ct. Oncogene. 2008 Nov 6;27(52):6667-78. doi: 10.1038/onc.2008.256. Epub 2008 Jul 28. Oncogene. 2008. PMID: 18663355

5

Loss of annexin A1 expression in breast cancer progression.

Cao Y, Li Y, Edelweiss M, Arun B, Rosen D, Resetkova E, Wu Y, Liu J, Sahin A, Albarracin CT. Cao Y, et al. Among authors: albarracin ct. Appl Immunohistochem Mol Morphol. 2008 Dec;16(6):530-4. doi: 10.1097/PAI.0b013e31817432c3. Appl Immunohistochem Mol Morphol. 2008. PMID: 18776816

6

Prognostic impact of discordance between triple-receptor measurements in primary and recurrent breast cancer.

Liedtke C, Broglio K, Moulder S, Hsu L, Kau SW, Symmans WF, Albarracin C, Meric-Bernstam F, Woodward W, Theriault RL, Kiesel L, Hortobagyi GN, Pusztai L, Gonzalez-Angulo AM. Liedtke C, et al. Ann Oncol. 2009 Dec;20(12):1953-8. doi: 10.1093/annonc/mdp263. Epub 2009 Jul 12. Ann Oncol. 2009. PMID: 19596702 Free PMC article.

7

Brk/PTK6 sustains activated EGFR signaling through inhibiting EGFR degradation and transactivating EGFR.

Li X, Lu Y, Liang K, Hsu JM, Albarracin C, Mills GB, Hung MC, Fan Z. Li X, et al. Oncogene. 2012 Oct 4;31(40):4372-83. doi: 10.1038/onc.2011.608. Epub 2012 Jan 9. Oncogene. 2012. PMID: 22231447 Free PMC article.

8

Biologic features and prognosis of ductal carcinoma in situ are not adversely impacted by initial large body mass.

Kuerer HM, Lari SA, Arun BK, Hu CY, Brewster A, Mittendorf EA, Albarracin CT, Babiera GV, Caudle AS, Wagner JL, Litton JK, Bedrosian I, Meric-Bernstam F, Lucci A, Hunt KK. Kuerer HM, et al. Among authors: albarracin ct. Breast Cancer Res Treat. 2012 Jun;133(3):1131-41. doi: 10.1007/s10549-012-1999-3. Epub 2012 Mar 4. Breast Cancer Res Treat. 2012. PMID: 22392043 Free PMC article.

9

Invasive paget disease of the breast: clinicopathologic study of an underrecognized entity in the breast.

Duan X, Sneige N, Gullett AE, Prieto VG, Resetkova E, Andino LM, Wu Y, Gilcrease MZ, Bedrosian I, Dawood S, Arun B, Albarracin CT. Duan X, et al. Among authors: albarracin ct. Am J Surg Pathol. 2012 Sep;36(9):1353-8. doi: 10.1097/PAS.0b013e318259ef7f. Am J Surg Pathol. 2012. PMID: 22895267

10

Suppression of Akt-mTOR pathway-a novel component of oncogene induced DNA damage response barrier in breast tumorigenesis.

Bhardwaj A, Rosen D, Liu M, Liu Y, Hao Q, Ganesan N, Etzel CJ, Gullett A, Albarracin CT, Bedrosian I. Bhardwaj A, et al. Among authors: albarracin ct. PLoS One. 2014 May 8;9(5):e97076. doi: 10.1371/journal.pone.0097076. eCollection 2014. PLoS One. 2014. PMID: 24811059 Free PMC article.

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