Lin S - Search Results

1

The neuromuscular junction is a focal point of mTORC1 signaling in sarcopenia.

Ham DJ, Börsch A, Lin S, Thürkauf M, Weihrauch M, Reinhard JR, Delezie J, Battilana F, Wang X, Kaiser MS, Guridi M, Sinnreich M, Rich MM, Mittal N, Tintignac LA, Handschin C, Zavolan M, Rüegg MA. Ham DJ, et al. Among authors: lin s. Nat Commun. 2020 Sep 9;11(1):4510. doi: 10.1038/s41467-020-18140-1. Nat Commun. 2020. PMID: 32908143 Free PMC article.

2

An agrin minigene rescues dystrophic symptoms in a mouse model for congenital muscular dystrophy.

Moll J, Barzaghi P, Lin S, Bezakova G, Lochmüller H, Engvall E, Müller U, Ruegg MA. Moll J, et al. Among authors: lin s. Nature. 2001 Sep 20;413(6853):302-7. doi: 10.1038/35095054. Nature. 2001. PMID: 11565031

3

Mammalian TOR complex 2 controls the actin cytoskeleton and is rapamycin insensitive.

Jacinto E, Loewith R, Schmidt A, Lin S, Rüegg MA, Hall A, Hall MN. Jacinto E, et al. Among authors: lin s. Nat Cell Biol. 2004 Nov;6(11):1122-8. doi: 10.1038/ncb1183. Epub 2004 Oct 3. Nat Cell Biol. 2004. PMID: 15467718

4

Overexpression of mini-agrin in skeletal muscle increases muscle integrity and regenerative capacity in laminin-alpha2-deficient mice.

Bentzinger CF, Barzaghi P, Lin S, Ruegg MA. Bentzinger CF, et al. Among authors: lin s. FASEB J. 2005 Jun;19(8):934-42. doi: 10.1096/fj.04-3376com. FASEB J. 2005. PMID: 15923403

5

Src-family kinases stabilize the neuromuscular synapse in vivo via protein interactions, phosphorylation, and cytoskeletal linkage of acetylcholine receptors.

Sadasivam G, Willmann R, Lin S, Erb-Vögtli S, Kong XC, Rüegg MA, Fuhrer C. Sadasivam G, et al. Among authors: lin s. J Neurosci. 2005 Nov 9;25(45):10479-93. doi: 10.1523/JNEUROSCI.2103-05.2005. J Neurosci. 2005. PMID: 16280586 Free PMC article.

6

Linker molecules between laminins and dystroglycan ameliorate laminin-alpha2-deficient muscular dystrophy at all disease stages.

Meinen S, Barzaghi P, Lin S, Lochmüller H, Ruegg MA. Meinen S, et al. Among authors: lin s. J Cell Biol. 2007 Mar 26;176(7):979-93. doi: 10.1083/jcb.200611152. J Cell Biol. 2007. PMID: 17389231 Free PMC article.

We have previously shown in mouse models for MDC1A that muscle-specific overexpression of a miniaturized form of agrin (mini-agrin), which binds to dystroglycan but not to alpha7beta1 integrin, substantially ameliorates the disease (Moll, J., P. Barzaghi, S. Lin, G. …

We have previously shown in mouse models for MDC1A that muscle-specific overexpression of a miniaturized form of agrin (mini-agrin), which b …

7

Identification of a lectin causing the degeneration of neuronal processes using engineered embryonic stem cells.

Plachta N, Annaheim C, Bissière S, Lin S, Rüegg M, Hoving S, Müller D, Poirier F, Bibel M, Barde YA. Plachta N, et al. Among authors: lin s. Nat Neurosci. 2007 Jun;10(6):712-9. doi: 10.1038/nn1897. Epub 2007 May 7. Nat Neurosci. 2007. PMID: 17486104

8

Tyrosine phosphatases such as SHP-2 act in a balance with Src-family kinases in stabilization of postsynaptic clusters of acetylcholine receptors.

Camilleri AA, Willmann R, Sadasivam G, Lin S, Rüegg MA, Gesemann M, Fuhrer C. Camilleri AA, et al. Among authors: lin s. BMC Neurosci. 2007 Jul 2;8:46. doi: 10.1186/1471-2202-8-46. BMC Neurosci. 2007. PMID: 17605785 Free PMC article.

9

Synapse loss in cortex of agrin-deficient mice after genetic rescue of perinatal death.

Ksiazek I, Burkhardt C, Lin S, Seddik R, Maj M, Bezakova G, Jucker M, Arber S, Caroni P, Sanes JR, Bettler B, Ruegg MA. Ksiazek I, et al. Among authors: lin s. J Neurosci. 2007 Jul 4;27(27):7183-95. doi: 10.1523/JNEUROSCI.1609-07.2007. J Neurosci. 2007. PMID: 17611272 Free PMC article.

10

The role of nerve- versus muscle-derived factors in mammalian neuromuscular junction formation.

Lin S, Landmann L, Ruegg MA, Brenner HR. Lin S, et al. J Neurosci. 2008 Mar 26;28(13):3333-40. doi: 10.1523/JNEUROSCI.5590-07.2008. J Neurosci. 2008. PMID: 18367600 Free PMC article.

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